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January 31, 2017 – 1:38 pm | 1,880 views

What Works For Anxiety Disorders–Anti-Anxiety Drugs

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Antidepressant Drugs »»» Celexa (citalopram) Luvox (fluvoxamine) Paxil (paroxetine) Prozac (fluoxetine) Zoloft (sertraline)


Antipsychotic Drugs »»» Moban Risperdal (risperidone) Seroquel (quetiapine) Stelazine Zyprexa (olanzapine)

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Mood Stabalizers »»» Depakene Depakote sprinkles Lamictal (lamatrogine) Lithium (lithium carbonate) Eskalith Lithobid


Antianxiety Drugs »»» Ativan (lorazepam) BuSpar (buspirone) Klonopin (clonazepam) Valium (diazepam) Xanax (alprazolam)

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Anti-epileptic drugs (AEDs) are the main form of treatment for people with epilepsy. There are around 26 AEDs used to treat seizures.

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Why do people have withdrawal problems?

Submitted by on August 21, 2016 – 11:36 am | 146 views

Why do people have ?

Most psychiatric drugs work on body chemicals called neurotransmitters, which carry messages between , or between nerves and , or other organs. They cross the gap (the synapse) between the nerve and its target. There are a lot of different neurotransmitters, and in the brain they interact and influence each other in subtle ways, which we have scarcely begun to understand. The main neurotransmitters, about which we know most, are dopamine, noradrenaline

(norepinephrine), (5-hydroxytryptamine or ) and . Psychiatric drugs may have various actions:

  • aim to raise the levels of noradrenaline or serotonin in the brain
  • reduce the effects of dopamine
  • Drugs for dementia aim to increase levels of acetylcholine
  • Mood stabilisers don’t actually target neurotransmitters directly, but affect brain activity by different mechanisms (see p. 20 for more information).

The neurotransmitters work by interacting with receptors. These are minute areas on the nerve or other cells that make the cell respond in the appropriate way. Some psychiatric drugs block these receptors, reducing the effect of a neurotransmitter. Others increase the level of a neurotransmitter, so its effects last longer. Because they all interact, changing the level of one neurotransmitter will change others; so however well a drug is targeted towards a particular receptor, it will have a knock-on effect on the whole system.

One knock-on effect of many psychiatric drugs is to suppress acetylcholine, and this causes some of the side effects people experience. Using a drug can change the number of receptors people have, and this seems to be the cause of many withdrawal problems:

  • If a drug increases the levels of neurotransmitters at the synapse, then the number of receptors there reduces. This means the person may not have enough receptors after the drug is withdrawn, making them more likely to relapse. ( will raise the concentration of serotonin by inhibiting its reuptake.)
  • If a drug blocks the receptors, then new receptors are created to make up the shortfall. This makes the person extra sensitive to the neurotransmitter, so that when he or she comes off the drug, this ‘supersensitivity’ makes them more vulnerable to psychosis and at a high risk of suffering a relapse. (Antipsychotics block .) Newer ‘atypical’ antipsychotic drugs are better targeted to particular dopamine receptors, but they still have serious side effects and should be limited to the lowest possible dose for the shortest possible time. A lot of people who have been ‘stuck’ for years on the older drugs might do better on the newer drugs, or might at least like to try.

Source: Making sense of coming off psychiatric drugs

A previous article entitled Total Number of People Taking Psychiatric Drugs in the United States provides information... ADHD, All Psychiatric Drugs ve Anti-anxiety

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